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Resident Manual of Trauma to the Face, Head, and Neck

224

Chapter 10: Foreign Bodies and Caustic Ingestion

2. Pulmonary

a. Atelectasis

Atelectasis is usually asymptomatic and will resolve with patient

mobility or incentive spirometer.

b. Pneumonia

Pneumonia may be the presenting symptom or may develop a few days

following removal of the foreign body. Treat with antibiotic coverage as

noted in I.F, above. If the patient’s symptoms do not improve with

adequate therapy, a missed second foreign body should be considered.

c. Bleeding

Bleeding is usually from mucosal trauma or granulation tissue. This is

often self-limiting. In rare instances, vessel erosion may lead to a

significant bleed, requiring urgent thoracic surgery intervention.

d. Pneumothorax or Pneumomediastinum

Pneumothorax or pneumomediastinum is usually from a small perfora-

tion in the airway that heals spontaneously and does not require further

intervention. Symptomatic patients may be treated with oxygen and

serial chest x-rays. If the pneumothorax or pneumomediastinum

increases in size or is large on initial identification, a thoracic surgery

consult should be called for further intervention.

3. Esophageal

a. Bleeding

Bleeding is usually from direct mucosal trauma and is self-limiting.

Consider proton pump inhibitors and/or H2 blockers to prevent further

injury to the damaged mucosa.

b. Esophageal Perforation

y

y

Early recognition and management of esophageal perforations have

decreased the mortality rate from 60 percent to 9 percent from

complications, such as a retroesophageal abscess or mediastinitis.

y

y

Cervical subcutaneous emphysema, fever, tachycardia, tachypnea,

and increased pain may all be early signs of a perforation.

y

y

If a small esophageal perforation is suspected intraoperatively, place

a nasogastric tube and perform a barium swallow or gastrografin

study. If esophageal perforation is confirmed, keep the patient NPO,

and consider broad-spectrum antibiotics.

III. Caustic Ingestion

The incidence of caustic ingestion has decreased since the Federal

Hazardous Substances Act of 1960 and the Poison Prevention